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PDR001

Phase 2

Melanoma | Monoclonal antibody | Oncology |Novartis AG|Last Updated: Jul 28, 2026

Success Probability

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Market & Valuation

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Trial Design

RandomizedCONTROLLEDDMC
Total Trials2
Total Enrollment515

FDA Designations

No designations recorded

Clinical trial landscape

PDR001 · 11 trials · 31 indications

Phase 2 3Phase 1 8
NCT03484923Study of Efficacy and Safety of Novel Spartalizumab Combinations in Patients With Previously Treated Unresectable or Metastatic MelanomaMelanoma
COMPLETED196 Analytics
NCT03365791PDR001 Plus LAG525 for Patients With Advanced Solid and Hematologic MalignanciesSmall Cell Lung Cancer
COMPLETED76 Analytics
NCT02955069Study of Efficacy and Safety of PDR001 in Patients With Advanced or Metastatic, Well-differentiated, Non-functional Neuroendocrine Tumors of Pancreatic, Gastrointestinal (GI), or Thoracic Origin or Poorly-differentiated Gastroenteropancreatic Neuroendocrine Carcinoma (GEP-NEC)Well-differentiated Non-functional NET of Thoracic Origin
COMPLETED116 Analytics
PHASE2COMPLETED
Study of Efficacy and Safety of Novel Spartalizumab Combinations in Patients With Previously Treated Unresectable or Metastatic Melanoma
MelanomaUnlock trial analytics
PHASE2COMPLETED
PDR001 Plus LAG525 for Patients With Advanced Solid and Hematologic Malignancies
Small Cell Lung CancerUnlock trial analytics
PHASE2COMPLETED
Study of Efficacy and Safety of PDR001 in Patients With Advanced or Metastatic, Well-differentiated, Non-functional Neuroendocrine Tumors of Pancreatic, Gastrointestinal (GI), or Thoracic Origin or Poorly-differentiated Gastroenteropancreatic Neuroendocrine Carcinoma (GEP-NEC)
Well-differentiated Non-functional NET of Thoracic OriginUnlock trial analytics

Study Endpoints

Primary Endpoints

Overall Response Rate (ORR)
Up to 49 months (randomized section) and 18 months (non-randomized section)

ORR defined as the percentage of patients with a best overall response of either confirmed complete response (CR) or partial response (PR) as per local review by Response Evaluation Criteria In Solid Tumors Criteria (RECIST v1.1) and assessed by computed tomography (CT)/ magnetic resonance imaging (MRI). CR:Disappearance of all non-nodal target and non-target lesions. In addition, any pathological lymph nodes assigned as target lesions must have a reduction in short axis to \< 10 mm PR: At least a 30% decrease in the sum of diameter of all target lesions, taking as reference the baseline sum of diameters.

Clinical Benefit Rate (CBR) at 24 Weeks of PDR001+LAG525 by Tumor Type in Multiple Solid Tumors and Lymphoma
24 weeks

CBR is defined as the percentage of participants with a best overall response of Complete Response (CR), Partial Response (PR) and Stable Disease (SD). Tumor response was based on local investigator assessment. For participants with solid tumors the assessment criteria was Response Evaluation Criteria in Solid Tumors version 1.1 (RECIST v1.1) and for participants with lymphoma the assessment criteria was the Revised Response Criteria for Malignant Lymphoma (Cheson et al 2007). For RECIST v1.1, CR=Disappearance of all non-nodal target lesions. In addition, any pathological lymph nodes assigned as target lesions must have a reduction in short axis to \< 10 mm; PR= At least a 30% decrease in the sum of diameter of all target lesions, taking as reference the baseline sum of diameters; SD= Neither sufficient shrinkage to qualify for PR or CR nor an increase in lesions which would qualify for progression. CBR (CR+PR+SD) is reported overall and by tumor type.

Overall Response Rate (ORR) by RECIST 1.1 and as Per Blinded Independent Central Review (BIRC).
From baseline up to approximately 1.5 years

ORR is defined as the proportion of patients with best overall response (BOR) of complete response (CR) or partial response (PR), according to BIRC radiological assessment by RECIST 1.1. CR: disappearance of all non-nodal target lesions. In addition, any pathological lymph nodes assigned as target lesions must have a reduction in short axis to \< 10 mm. PR: at least a 30% decrease in the sum of diameter of all target lesions, taking as reference the baseline sum of diameters.

Frequency and nature of AE and SAE by subject
5 years

Safety data.

Number of subjects with PDR001 dose interruption and/or reduction
5 years
Number of patients reporting dose limiting toxicities
2 months

number of patients reporting dose limiting toxicity

The number of patients who experience a treatment-related adverse event after being treated with a single dose of single agent CJM112, or two doses of PDR001 in combination with CJM112 or LCL161
24 months

Number of patients with treatment-related adverse events as assessed by CTCAE v4.0

The number of patients requiring interruptions after a single dose of single agent CJM112, or two doses of PDR001 in combination with CJM112 or LCL161
24 months

Frequency of patients requiring a dose interruption

The number of patients treated with single agent CJM112, or PDR001 in combination with either CJM112 or LCL161, who discontinued treatment
24 months

Frequency of patients discontinuing treatment.

The number of patients requiring a dose reduction after a single dose of single agent CJM112, or two doses of PDR001 in combination with CJM112 or LCL161
24 months

Frequency of patients requiring a dose reduction.

Safety of MBG453 single agent treatment or MBG453 in combination with PDR001 or PDR001 and/or MBG453 in combination with decitabine or azacitidine.
24 months

Incidence and severity of AEs and SAEs

Incidence of Dose Limiting Toxicities (DLTs)
2 months

The incidence of DLTs during the first two cycles of treatment with MBG453 in combination with PDR001 or PDR001 and/or MBG453 in combination with decitabine.

Tolerability of MBG453 single agent treatment or MBG453 in combination with PDR001 or PDR001 and/or MBG453 in combination with decitabine or azacitidine.
24 months

Incidence and severity of AEs and SAEs

Number of participants with Adverse Events (AEs) and Serious Adverse Events (SAEs)
From baseline until 30 days of last dose of study treatment

Incidence and severity of AEs and SAEs, including changes in laboratory vital signs and ECGs

Incidendence of Dose Limiting Toxicities (DLTs)
During the first 8 weeks of treatment

A dose-limiting toxicity (DLT) was defined as an adverse event or abnormal laboratory value assessed as unrelated to disease progression, inter-current illness, or concomitant medications that met certain criteria as defined in the protocol.

Dose interruptions
Until end of treatment, assessed for a median time of 4 months

Tolerability measured by the number of subjects who have interruptions of study treatment

Dose reductions
Until end of treatment, assessed for a median time of 4 months

Tolerability measured by the number of subjects who have reductions of study treatment

Dose intensity
Until end of treatment, assessed for a median time of 4 months

Tolerability measured by the dose intensity of study treatment

Phase 1: Incidence of dose limiting toxicities (DLTs)
5.5 years

During the first two cycles Cycle = 28 days

Frequency of dose interruptions and reductions
5.5 years

Through study completion, an average of 6 months

Frequency and severity of treatment-emergent adverse events (AEs) and serious adverse events (SAEs)
6 years

Through study completion, an average of 6 months

Changes between baseline and post-baseline laboratory parameters and vital signs
6 years

Through study completion, an average of 6 months

Dose intensities
6 years

Through study completion, an average of 6 months

Frequency of treatment-emergent Adverse Events (AEs) and Serious Adverse Events (SAEs) as a measure of safety
Throughout the study at every visit, an average of 1 year
Changes between baseline and post-baseline laboratory parameters and vital signs.
Baseline and throughout the study at every visit, an average of 1 year
Incidence of dose limiting toxicities (DLTs) of treatment (Escalation only)
During the first two cycles; Cycle = 28 days
Frequency of dose interruptions
Throughout the study at every visit, an average of 1 year
Severity of treatment-emergent Adverse Events (AEs) and Serious Adverse Events (SAEs) as a measure of safety
Throughout the study at every visit, an average of 1 year
Frequency of dose reductions
Throughout the study at every visit, an average of 1 year
Phase l: The Exposure (AUC(0-336h)) After First Dose of Treatment at Cycle 3 (Each Cycle = 28 Days)
Predose, 1hour (h), 24h, 48h, 72h, 168h, 240h, 336h post dose (cycle 3)

Estimated the recommended phase 2 dose (RP2D) and/or the maximum tolerated dose (MTD) for PDR001. AUC0-336h is the AUC from time zero to 336 hour post dose of a measurable concentration sampling time.

Phase l: Incidence of Dose Limiting Toxicities (DLTs)
8 months

DLT is defined as an adverse event (AE) or abnormal laboratory value of common terminology criteria for adverse events (CTCAE) grade ≥ 3 assessed as unrelated to disease, disease progression, inter-current illness or concomitant medications, which occurs within the first cycle of treatment with PDR001 during the dose escalation part of the study for which relationship to study treatment cannot be ruled out, with some exceptions.

Phase ll: Overall Response Rate (ORR) Per Response Evaluation Criteria in Solid Tumors (RECIST v1.1)
61 months

ORR is the percentage of participants with a best overall response of complete response (CR) or partial response (PR) as per Response Evaluation Criteria in Solid Tumors (RECIST) v1.1 CR = at least 2 determinations of CR at least 4 weeks apart before progression where confirmation required or 1 determination of CR prior to progression where confirmation not required. PR = at least 2 determinations of PR or better at least 4 weeks apart before progression (and not qualifying for a CR) where confirmation required or 1 determination of PR prior to progression where confirmation not required. RECIST criteria is a set of published rules that define when tumors in cancer patients improve ("respond"), stay the same ("stabilize"), or worsen ("progress") during treatment.

Secondary Endpoints

Duration of Response (DOR)
From first documented response to disease progression or death due to any cause, whichever occurs first, assessed up to 49 months (randomized part) and 18 months (non-randomized part)
Overall Survival (OS)
From randomization (or start of treatment for non-randomized section) to death due to any cause, assessed up to 49 months (randomized section) and 24 months (non-randomized section)
Progression Free Survival (PFS)
From randomization (or start of treatment for non-randomized section) to disease progression or death due to any cause, whichever occurs first, assessed up to 49 months (randomized section) and 18 months (non-randomized section)
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Study Design & Arms

AllocationRANDOMIZED
MaskingNONE
ModelPARALLEL
PurposeTREATMENT

Treatment Arms

ArmTypeDescription
Arm 1: LAG525 + PDR001 (randomized section)EXPERIMENTALParticipnats randomized to receive LAG525 at a dosage of 600 mg administered intravenously every 4 weeks, in combination with PDR001 at a dosage of 400 mg administered intravenously every 4 weeks
Arm 2: INC280+PDR001 (randomized section)EXPERIMENTALParticipants randomized to receive INC280 orally at a dosage of 400 mg twice daily, in combination with PDR001 intravenously at a dosage of 400 mg every 4 weeks
Arm 3: ACZ885 + PDR001 (randomized section)EXPERIMENTALParticipants randomized to receive to receive ACZ885 at a dosage of 300 mg administered subcutaneously every 4 weeks, in combination with PDR001 at a dosage of 400 mg administered intravenously every 4 weeks
Arm 4: LEE011 + PDR001 (randomized section)EXPERIMENTALParticipants randomized to receive LEE011 orally at a dosage of 600 mg once daily on Days 1-21 of a 28-day cycle, in combination with PDR001 intravenously at a dosage of 400 mg every 4 weeks
Arm 1A: LAG525 + PDR001 (non-randomized section)EXPERIMENTALLAG-3 positive participants received LAG525 at a dosage of 600 mg administered intravenously every 4 weeks, in combination with PDR001 at a dosage of 400 mg administered intravenously every 4 weeks
PDR001+LAG525EXPERIMENTALPDR001 300 mg and LAG525 400 mg administered via i.v. infusion over 30 minutes once every 3 weeks (Q3W). LAG525 was given first followed by PDR001.
PDR001EXPERIMENTALSubjects with advanced or metastatic, well-differentiated, NET of pancreatic, GI, or thoracic origin or poorly-differentiated GEP-NEC, that have progressed on prior treatment were treated with 400mg PDR001 administered via intravenous infusion once every 4 weeks.
Arm AEXPERIMENTALDose escalation of single agent CJM112
Arm BEXPERIMENTALDose escalation of CJM112 in combination with a fixed dose of PDR001
Arm CEXPERIMENTALDose escalation of LCL161 in combination with a fixed dose of PDR001
Decitabine and PDR001EXPERIMENTALDecitabine in combination with PDR001
Decitabine and MBG453EXPERIMENTALDecitabine in combination with MBG453
Decitabine, PDR001 and MBG453EXPERIMENTALDecitabine in combination with PDR001 and MBG453
MBG453EXPERIMENTALMBG453 alone
MBG453 and PDR001EXPERIMENTALMBG453 in combination with PDR001
Azacitidine and MBG453EXPERIMENTALAzacitidine in combination with MBG453
PDR001 + SorafenibOTHERPDR001 at 400 mg given intravenously every 4 weeks and sorafenib 400 mg taken orally once or twice per day (escalating doses)
CRC - PDR001 + LCL161EXPERIMENTALEnrollment to this combination arm is closed to further enrollment.
NSCLC - PDR001 + LCL161EXPERIMENTALEnrollment to this combination arm is closed to further enrollment.
TNBC - PDR001 + LCL161EXPERIMENTALEnrollment to this combination arm is closed to further enrollment.
CRC - PDR001+ EverolimusEXPERIMENTALEnrollment to this combination arm is closed to further enrollment.
NSCLC - PDR001+ EverolimusEXPERIMENTALEnrollment to this combination arm is closed to further enrollment.
TNBC - PDR001+ EverolimusEXPERIMENTALEnrollment to this combination arm is closed to further enrollment.
CRC - PDR001 + PanobinostatEXPERIMENTALEnrollment to this combination arm is closed to further enrollment.
NSCLC - PDR001 + PanobinostatEXPERIMENTALEnrollment to this combination arm is closed to further enrollment.
TNBC - PDR001 + PanobinostatEXPERIMENTALEnrollment to this combination arm is closed to further enrollment.
CRC - PDR001 + QBM076EXPERIMENTALEnrollment to this combination arm is closed to further enrollment.
TNBC - PDR001 + QBM076EXPERIMENTALEnrollment to this combination arm is closed to further enrollment.
NSCLC- PDR001 + QBM076EXPERIMENTALEnrollment to this combination arm is closed to further enrollment.
CRC - PDR001 + HDM201EXPERIMENTALDose escalation completed, expansion arm.
RCC - PDR001 + HDM201EXPERIMENTALDose escalation completed, expansion arm.
PDR + ACZ 100mg Q8WEXPERIMENTALPDR + ACZ 100mg Q8W
PDR + ACZ 300mg Q8WEXPERIMENTALPDR + ACZ 300mg Q8W
PDR + ACZ RDE TNBCEXPERIMENTALPDR + ACZ Recommended Dose for Expansion (RDE) Triple Negative Breast Cancer (TNBC)
PDR + ACZ RDE NSCLCEXPERIMENTALPDR + ACZ Recommended Dose for Expansion (RDE) Non-Small Cell Lung Cancer (NSCLC)
PDR + ACZ RDE CRCEXPERIMENTALPDR + ACZ Recommended Dose for Expansion (RDE) Colorectal Cancer (CRC)
PDR + CJM 25mg Q4WEXPERIMENTALPDR + CJM 25mg Q4W
PDR + CJM 75mg Q4WEXPERIMENTALPDR + CJM 75mg Q4W
PDR + CJM 225mg Q4WEXPERIMENTALPDR + CJM 225mg Q4W
PDR + CJM 450mg Q4WEXPERIMENTALPDR + CJM 450mg Q4W
PDR + CJM 450mg Q2WEXPERIMENTALPDR + CJM 450mg Q2W
PDR + CJM 900mg Q4WEXPERIMENTALPDR + CJM 900mg Q4W
PDR + CJM 900mg Q2WEXPERIMENTALPDR + CJM 900mg Q2W
PDR + CJM 1200mg Q4WEXPERIMENTALPDR + CJM 1200mg Q4W
PDR + TMT 0.5mg QDEXPERIMENTALPDR + TMT 0.5mg QD
PDR + TMT 1mg QDEXPERIMENTALPDR + TMT 1mg QD
PDR + TMT 1mg QD, 3 Weeks on/1 Week offEXPERIMENTALPDR + TMT 1mg QD, 3 Weeks on/1 Week off
PDR + TMT 1.5 mg QD, 2 Weeks on/2 Weeks offEXPERIMENTALPDR + TMT 1.5 mg QD, 2 Weeks on/2 Weeks off
PDR + TMT 1.5 mg QD, 3 Weeks on/1 Week offEXPERIMENTALPDR + TMT 1.5 mg QD, 3 Weeks on/1 Week off
PDR + EGF816 25mg QDEXPERIMENTALPDR + EGF816 25mg QD
PDR + EGF816 50mg QDEXPERIMENTALPDR + EGF816 50mg QD
s.a. ACZ RDE TNBCEXPERIMENTALSingle agent (s.a.) ACZ Recommended Dose for Expansion (RDE) Triple Negative Breast Cancer (TNBC)
s.a. ACZ RDE NSCLCEXPERIMENTALSingle agent (s.a.) ACZ Recommended Dose for Expansion (RDE) Non-Small Cell Lung Cancer (NSCLC)
s.a. ACZ RDE CRCEXPERIMENTALSingle agent (s.a.) ACZ Recommended Dose for Expansion (RDE) Colorectal Cancer (CRC)
patients with solid tumorsOTHERPhase I Dose escalation cohorts
Selected tumor typesOTHERPhase II expansion: Selected tumor types: melanoma, NSCLC, triple negative breast cancer, anaplastic thyroid cancer

Interventions

NameTypeDescription
PDR001DRUG400 mg of PDR001 administered every 4 weeks intravenously
LAG525DRUG600 mg of LAG525 administered every 4 weeks intravenously
INC280DRUG400 mg of INC280 administered twice daily orally
ACZ885DRUG200 mg of ACZ885 administered every 4 weeks subcutaneosuly
LEE011DRUG600 mg of LEE011 orally taken once daily on Days 1-21 of a 28-day cycle
CJM112DRUGAnti-IL-17A antibody
LCL161DRUGOral small molecule SMAC-mimetic
DecitabineDRUGDecitabine is a cytidine deoxynucleoside analogue that selectively inhibits DNA methyltransferases at low doses, resulting in gene promoter hypomethylation.
MBG453DRUGMBG453 is a high-affinity, humanized anti-TIM-3 IgG4 monoclonal antibody which blocks the binding of TIM-3 to phosphatidylserine (PtdSer).
AzacitidineDRUGAzacitidine (5-azacytidine) is a cytidine nucleoside analogue that selectively inhibits DNA methyltransferases at low doses, resulting in gene promoter hypomethylation
SorafenibDRUGSorafenib is formulated as a tablet.
EverolimusDRUG -
PanobinostatDRUG -
QBM076DRUG -
HDM201DRUG -
TMT212DRUGTablets
EGF816DRUGTablets
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Eligibility Criteria

Age Range18 Years to N/A
SexALL
Healthy VolunteersNo
Study Sites30

Key inclusion criteria for Arm 1, 2, 3, 4: * Histologically confirmed unresectable or metastatic stage IIIB/C/D or IV melanoma using AJCC edition 8. * Previously treated for unresectable or metastatic melanoma: * Subjects with V600BRAF wild-type disease had to have received prior systemic therap...

Countries:United StatesAustraliaCanadaFranceGermanyItalyNetherlandsSpainSwitzerlandUnited KingdomAustriaBelgiumJapanChinaCzechiaHong KongHungaryPolandSouth KoreaTaiwanThailandFinlandIsraelSingaporeLebanonNorwayTurkey (Türkiye)
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Recent Changes (Last 90 Days)

LOWJul 28, 2026NCT04058756lastUpdatePostDate: changed
LOWJul 28, 2026NCT04058756lastUpdatePostDate: changed

Frequently asked questions about PDR001

What is PDR001 used for?

PDR001 is an investigational small molecule being studied for advanced malignancies, including small cell lung cancer, well-differentiated non-functional NET of thoracic origin, melanoma, colorectal cancer, triple negative breast cancer, NSCLC adenocarcinoma, and advanced solid tumors. It is in Phase 1 clinical development.

Who makes PDR001?

PDR001 is being developed by Novartis AG, which trades under the ticker NVS. The drug is an investigational oncology small molecule currently in Phase 1 clinical trials.

What phase is PDR001 in?

PDR001 is in Phase 1 clinical development. It is an investigational drug, meaning it has not been approved by regulatory authorities and is still being studied in clinical trials for safety and efficacy.

What clinical trials is PDR001 in?

PDR001 has been studied in several Phase 1 trials, including NCT02678260 in advanced malignancies, NCT02900664 in combination with other agents for colorectal cancer, triple negative breast cancer, and NSCLC adenocarcinoma, NCT02988440 with sorafenib in hepatocellular carcinoma, and NCT03066648 with decitabine in AML or high risk MDS.

Is PDR001 the same as spartalizumab?

PDR001 is also known as spartalizumab. It is an investigational oncology drug being developed by Novartis AG for multiple cancer types, including melanoma, colorectal cancer, and advanced solid tumors.