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Alirocumab

Phase 3

Hypercholesterolemia | Monoclonal antibody | Metabolic |Regeneron Pharmaceuticals, Inc.|Last Updated: Jul 22, 2026

Target and mechanism

Molecular targetPCSK9/PD-1
ModalityMonoclonal antibody

Success Probability

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Market & Valuation

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Trial Design

RandomizedDouble-BlindPLACEBO_CONTROLLEDDMC
Total Trials8
Total Enrollment2,355

FDA Designations

No designations recorded

Clinical trial landscape

Alirocumab · 13 trials · 5 indications

Phase 3 7Phase 2 5Phase 1 1
NCT03156621Study in Participants With Homozygous Familial Hypercholesterolemia (HoFH)Homozygous Familial Hypercholesterolemia
COMPLETED69 Analytics
NCT02326220Study of Alirocumab (REGN727/SAR236553) in Patients With Heterozygous Familial Hypercholesterolemia (HeFH) Undergoing Low-density Lipoprotein (LDL) Apheresis TherapyHeterozygous Familial Hypercholesterolemia
COMPLETED62 Analytics
NCT01926782Study to Evaluate the Efficacy and Safety of an Every Four Weeks Treatment Regimen of Alirocumab (REGN727/ SAR236553) in Patients With Primary Hypercholesterolemia (ODYSSEY CHOICE 1)Hypercholesterolemia
COMPLETED803 Analytics
NCT01709500Study of Alirocumab (REGN727/SAR236553) in Patients With heFH (Heterozygous Familial Hypercholesterolemia) Who Are Not Adequately Controlled With Their LMT (Lipid-Modifying Therapy)Heterozygous Familial Hypercholesterolemia
COMPLETED249 Analytics
NCT01730053Study of Alirocumab (REGN727/SAR236553) added-on to Rosuvastatin Versus Other Lipid Modifying Treatments (LMT) (ODYSSEY OPTIONS II)Hypercholesterolemia
COMPLETED305 Analytics
NCT01730040Study of the Efficacy and Safety of Alirocumab (REGN727/SAR236553) in Combination With Other Lipid-modifying Treatment (LMT) (ODYSSEY OPTIONS I)Hypercholesterolemia
COMPLETED355 Analytics
NCT01709513Study of Alirocumab (REGN727/SAR236553) in Patients With Primary Hypercholesterolemia and Moderate, High, or Very High Cardiovascular (CV) Risk, Who Are Intolerant to Statins (ODYSSEY ALTERNATIVE)Hypercholesterolemia
COMPLETED314 Analytics
PHASE3COMPLETED
Study in Participants With Homozygous Familial Hypercholesterolemia (HoFH)
Homozygous Familial HypercholesterolemiaUnlock trial analytics
PHASE3COMPLETED
Study of Alirocumab (REGN727/SAR236553) in Patients With Heterozygous Familial Hypercholesterolemia (HeFH) Undergoing Low-density Lipoprotein (LDL) Apheresis Therapy
Heterozygous Familial HypercholesterolemiaUnlock trial analytics
PHASE3COMPLETED
Study to Evaluate the Efficacy and Safety of an Every Four Weeks Treatment Regimen of Alirocumab (REGN727/ SAR236553) in Patients With Primary Hypercholesterolemia (ODYSSEY CHOICE 1)
HypercholesterolemiaUnlock trial analytics
PHASE3COMPLETED
Study of Alirocumab (REGN727/SAR236553) in Patients With heFH (Heterozygous Familial Hypercholesterolemia) Who Are Not Adequately Controlled With Their LMT (Lipid-Modifying Therapy)
Heterozygous Familial HypercholesterolemiaUnlock trial analytics
PHASE3COMPLETED
Study of Alirocumab (REGN727/SAR236553) added-on to Rosuvastatin Versus Other Lipid Modifying Treatments (LMT) (ODYSSEY OPTIONS II)
HypercholesterolemiaUnlock trial analytics
PHASE3COMPLETED
Study of the Efficacy and Safety of Alirocumab (REGN727/SAR236553) in Combination With Other Lipid-modifying Treatment (LMT) (ODYSSEY OPTIONS I)
HypercholesterolemiaUnlock trial analytics
PHASE3COMPLETED
Study of Alirocumab (REGN727/SAR236553) in Patients With Primary Hypercholesterolemia and Moderate, High, or Very High Cardiovascular (CV) Risk, Who Are Intolerant to Statins (ODYSSEY ALTERNATIVE)
HypercholesterolemiaUnlock trial analytics

Study Endpoints

Primary Endpoints

Percent Change in Low-density Lipoprotein Cholesterol (LDL-C) From Baseline to Week 12 (Intent-to-Treat [ITT] Estimand)
Baseline to Week 12

The percent change in LDL-C from baseline to week 12 is defined as: 100x (LDL-C value at week 12 - LDL-C value at baseline) / LDL-C value at baseline.

Change in Standardized Rate of Apheresis Treatments From Week 7 to Week 18
Week 7 to Week 18 (before start of open-label treatment)

Rate of apheresis treatments were normalized by the number of planned apheresis treatments according to each participant's established schedule at screening, week -10 to week -2. The normalized rate of apheresis was defined for each participant as the number of actual apheresis treatments received from week 7 to week 18 divided by the number of planned apheresis treatments per randomization strata at baseline (6 for Q2W and 12 for QW).

Percent Change From Baseline in Calculated LDL-C in Participants Not Receiving Concomitant Statin Therapy - ITT Analysis
From Baseline to Week 24

Adjusted LS means and standard errors at Week 24 and at averaged Week 21 to 24 from MMRM including available post-baseline data from Week 4 to Week 24 regardless of status on- or off-treatment. ITT population (subjects without concomitant statin therapy): all randomized subjects who did not receive concomitant statin therapy, with one baseline and at least one post-baseline calculated LDL-C value on- or off-treatment. Alirocumab 75 mg Q2W arm (calibrator arm) was included only to facilitate comparison of results of this study with the results of other studies that used an alirocumab 75 mg Q2W regimen. Hence no statistical comparison was performed for this arm.

Percent Change From Baseline in Calculated LDL-C in Participants Receiving Concomitant Statin Therapy - Intent-to-Treat (ITT Analysis)
From Baseline to Week 24

Adjusted least squares (LS) means and standard errors at Week 24 and at averaged Week 21 to 24 were obtained from a mixed effect model with repeated measures (MMRM) model to account for missing data. All available post-baseline data from Week 4 to Week 24 regardless of status on- or off-treatment were used in this model (ITT analysis). ITT population (subjects with concomitant statin therapy): all randomized subjects who received concomitant statin therapy, with one baseline and at least one post-baseline calculated LDL-C value on- or off-treatment. Alirocumab 75 mg Q2W arm (calibrator arm) was included only to facilitate comparison of results of this study with the results of other studies that used an alirocumab 75 mg Q2W regimen. Hence no statistical comparison was performed for this arm.

Percent Change From Baseline in Calculated LDL-C at Week 24 - Intent--to--Treat (ITT) Analysis
From Baseline to Week 52

Calculated LDL-C values were obtained using the Friedewald formula. Adjusted Least- squares (LS) means and standard errors at Week 24 were obtained from a mixed -effect model with repeated measures (MMRM) to account for missing data. All available post -baseline data from Week 4 to Week 52 regardless of status on- or off-treatment were used in the model.

Percent Change From Baseline in Calculated LDL-C at Week 24 - Intent-to-Treat (ITT) Analysis
From Baseline to Week 24

Calculated LDL-C values were obtained from Friedewald formula. Adjusted Least-squares (LS) means and standard errors at Week 24 were obtained from a mixed-effect model with repeated measures (MMRM) to account for missing data. All available post-baseline data from Week 4 to Week 24 regardless of status on- or off-treatment were used in the model (ITT analysis).

Percent Change From Baseline in Calculated LDL-C at Week 24 - Intent--To-Treat (ITT) Analysis
From Baseline to Week 24

Calculated LDL-C values were obtained from Friedewald formula. Adjusted Least-squares (LS) means and standard errors at Week 24 were obtained from a mixed-effect model with repeated measures (MMRM) to account for missing data. All available post-baseline data from Week 4 to Week 24 regardless of status on- or off-treatment were used in the model (ITT analysis).

Response rate associated with combination of alirocumab and cemiplimab
Day 1 of treatment until the date of first documented progression or date of death, whichever comes first, assessed up to 110 weeks per RECIST 1.1

Ascertain the response rate associated with alirocumab and cemiplimab, with 95% confidence intervals. Response rate is defined as the proportion of treated subjects with a complete or partial response per RECIST 1.1 criteria. All patients who receive at least one dose of alirocumab and cemiplimab will be considered for the primary outcome analysis

Percent change in LDL-C
From Baseline through Week 12
Number of Participants Who Experienced Treatment Emergent Adverse Events (TEAEs), Serious TEAEs and TEAEs Leading to Death
Baseline (Day 1 of current study) to end of study (Week 218)

An AE was defined as any untoward medical occurrence in a participant administered a study drug which may or may not have a causal relationship with the study drug. Treatment- emergent adverse events (TEAEs) were defined as AEs that developed or worsened or became serious during on- treatment period (time from the first dose of study drug to the last dose of study drug plus 70 days). A serious adverse event (SAE) was defined as any untoward medical occurrence that resulted in any of the following outcomes: death, life- threatening, required initial or prolonged in-patient hospitalization, persistent or significant disability/incapacity, congenital anomaly/birth defect, or considered as medically important event. Any TEAE included participants with both serious and non-serious AEs.

Percent Change in Measured Serum Low-Density Lipoprotein Cholesterol (LDL-C) From Baseline to Day 15
Baseline to Day 15

By day 15, participants in groups A and C had received 1 subcutaneous (SC) dose of 150 mg alirocumab and participants in group B and D had received 1 SC dose of placebo. \[Baseline adjusted least squares (LS) means and standard errors were obtained using analysis of covariance (ANCOVA) model specifying the treatment arm as the fixed effect and the baseline measured LDL-C value as a covariate.\]

Percent Change From Baseline in Calculated LDL-C at Week 12 - On-treatment Analysis
From Baseline to Week 12 (LOCF)

Calculated LDL-C values were obtained using the Friedewald formula. Baseline adjusted least squares (LS) means and standard errors were estimated using an analysis of covariance (ANCOVA) model including available post-baseline data on treatment from first investigational medicinal product (IMP) injection up to 21 days after last IMP injection (on-treatment analysis). Missing Week 12 data were imputed by last observation carried forward \[LOCF\] method.

Bacterial endotoxin level
Hour 120

Levels of bacterial endotoxin will be compared between study arms.

Lipoteichoic acid level
Hour 120

Levels of lipoteichoic acid will be compared between study arms.

Secondary Endpoints

Percent Change in Apolipoprotein (Apo) B From Baseline to Week 12 (ITT Estimand)
Baseline to Week 12
Percent Change in Non-high-density Lipoprotein Cholesterol (Non-HDL-C) From Baseline to Week 12
Baseline to Week 12
Percent Change in Total Cholesterol (TC) From Baseline to Week 12
Baseline to Week 12
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Study Design & Arms

AllocationRANDOMIZED
MaskingTRIPLE
ModelPARALLEL
PurposeTREATMENT

Treatment Arms

ArmTypeDescription
Alirocumab SC Q2WEXPERIMENTALAlirocumab SC every 2 weeks (Q2W) from baseline (day 1) through week 10 during the double-blind treatment period Starting at week 12, and continuing through week 22, participants will receive open-label alirocumab SC Q2W
Placebo SC Q2WEXPERIMENTALMatching placebo SC Q2W from baseline through week 10 during the double-blind treatment period Starting at week 12, and continuing through week 22, participants will receive open-label alirocumab SC Q2W
Placebo Q2W (Double Blind Period)EXPERIMENTALPlacebo (for alirocumab) subcutaneous (SC) injection Q2W up to Week 16.
Alirocumab 150 mg Q2W (Double Blind Period)EXPERIMENTALAlirocumab 150 mg SC injection Q2W up to Week 16.
Alirocumab 150 Q2W (Open Label Treatment Period)EXPERIMENTALAlirocumab 150 mg SC injection Q2W starting from Week 18 up to Week 76.
Placebo Q2WPLACEBO_COMPARATORTwo subcutaneous (SC) injections of placebo (for alirocumab) Q2W with or without stable statin therapy for 48 weeks.
Alirocumab 75 mg/ Up 150 mg Q2WEXPERIMENTALOne SC injection of each Alirocumab 75 mg and placebo Q2W with or without stable statin therapy for 48 weeks. Alirocumab dose up-titrated to 150 mg from Week 12 when LDL-C levels ≥ 70 mg/dL (1.81 mmol/L) (for very high CV risk participants) or ≥ 100 mg/dL (2.59 mmol/L) (for moderate and high CV risk participants) at Week 8.
Alirocumab 300 mg/ Up 150 mg Q4WEXPERIMENTALTwo SC injections of Alirocumab 150 mg Q4W alternating with two SC injections of placebo Q4W with or without stable statin therapy for 48 weeks. Alirocumab dose up-titrated to 150 mg from Week 12 when LDL-C levels ≥ 70 mg/dL (1.81 mmol/L) (for very high CV risk participants) or ≥ 100 mg/dL (2.59 mmol/L) (for moderate and high CV risk participants) at Week 8.
Alirocumab 75 mg/up to 150 mgEXPERIMENTALAlirocumab 75 mg every two weeks (Q2W) added to stable dose of statin with or without LMT for 78 weeks. Alirocumab dose up-titrated to 150 mg Q2W from Week 12 when LDL-C levels ≥70 mg/dL at Week 8.
PlaceboPLACEBO_COMPARATORPlacebo matched to alirocumab SC injection for 78-week treatment duration.
Rosuvastatin 20 mgACTIVE_COMPARATORParticipants, who were receiving rosuvastatin 10 mg over-encapsulated tablet orally at baseline, received rosuvastatin 20 mg over-encapsulated tablet orally once daily (QD), placebo for alirocumab SC injection every two weeks (Q2W), and placebo for ezetimibe over-encapsulated tablet orally QD added to stable Lipid-Modifying Therapy (LMT) for 24 weeks.
Ezetimibe 10 mg + Rosuvastatin 10 mgACTIVE_COMPARATORParticipants, who were receiving rosuvastatin 10 mg over-encapsulated tablet orally at baseline, received ezetimibe 10 mg over-encapsulated tablet orally QD, rosuvastatin 10 mg over-encapsulated tablet orally QD, and placebo for alirocumab SC injection Q2W added to stable LMT for 24 weeks.
Alirocumab 75 mg/up to 150 mg + Rosuvastatin 10 mgEXPERIMENTALParticipants, who were receiving rosuvastatin 10 mg over-encapsulated tablet orally at baseline, received alirocumab 75 mg SC injection Q2W, rosuvastatin 10 mg over-encapsulated tablet orally QD, and placebo for ezetimibe over-encapsulated tablet orally QD added to stable LMT for 24 weeks. Alirocumab dose up-titrated to 150 mg Q2W from Week 12 when LDL-C levels ≥70 mg/dL (1.81 mmol/L) or ≥100 mg/dL (2.59 mmol/L) at Week 8, based on baseline disease characteristic and medical history.
Rosuvastatin 40 mgACTIVE_COMPARATORParticipants, who were receiving rosuvastatin 20 mg over-encapsulated tablet orally at baseline, received rosuvastatin 40 mg over-encapsulated tablet orally QD, placebo for alirocumab Q2W SC injection, and placebo for ezetimibe QD over-encapsulated tablet orally added to stable LMT for 24 weeks.
Ezetimibe 10 mg + Rosuvastatin 20 mgACTIVE_COMPARATORParticipants, who were receiving rosuvastatin 20 mg over-encapsulated tablet orally at baseline, received ezetimibe 10 mg over-encapsulated tablet orally QD, rosuvastatin 20 mg over-encapsulated tablet orally QD, and placebo for alirocumab Q2W SC injection added to stable LMT for 24 weeks.
Alirocumab 75 mg/ up to 150 mg + Rosuvastatin 20 mgEXPERIMENTALParticipants, who were receiving rosuvastatin 20 mg over-encapsulated tablet orally at baseline, received alirocumab 75 mg Q2W SC injection, rosuvastatin 20 mg over-encapsulated tablet orally QD, and placebo for ezetimibe over-encapsulated tablet orally QD added to stable LMT for 24 weeks. Alirocumab dose up-titrated to 150 mg Q2W from Week 12 when LDL-C levels ≥70 mg/dL (1.81 mmol/L) or ≥100 mg/dL (2.59 mmol/L) at Week 8, based on baseline disease characteristic and medical history.
Atorvastatin 40 mgACTIVE_COMPARATORParticipants, who were receiving atorvastatin 20 mg over-encapsulated tablets orally at baseline, received atorvastatin 40 mg over-encapsulated tablets orally once daily (QD), placebo for alirocumab SC injection every two weeks (Q2W), and placebo for ezetimibe over-encapsulated tablets orally QD added to stable Lipid-Modifying Therapy (LMT) for 24 weeks.
Ezetimibe 10 mg + Atorvastatin 20 mgACTIVE_COMPARATORParticipants, who were receiving atorvastatin 20 mg over-encapsulated tablets orally at baseline, received ezetimibe 10 mg over-encapsulated tablets orally QD, atorvastatin 20 mg over-encapsulated tablets orally QD, and placebo for alirocumab SC injection Q2W added to stable LMT for 24 weeks.
Alirocumab 75 mg/up to 150 mg + Atorvastatin 20 mgEXPERIMENTALParticipants, who were receiving atorvastatin 20 mg over-encapsulated tablets orally at baseline, received Alirocumab 75 mg SC injection Q2W, atorvastatin 20 mg over-encapsulated tablets orally QD, and placebo for ezetimibe over-encapsulated tablets orally QD added to stable LMT for 24 weeks. Alirocumab dose up-titrated to 150 mg Q2W from Week 12 when LDL-C levels ≥70 mg/dL (1.81 mmol/L) or ≥100 mg/dL (2.59 mmol/L) at Week 8, based on baseline disease characteristic and medical history.
Atorvastatin 80 mgACTIVE_COMPARATORParticipants, who were receiving atorvastatin 40 mg over-encapsulated tablets orally at baseline, received Atorvastatin 80 mg over-encapsulated tablets orally QD, placebo for alirocumab SC injection Q2W, and placebo for ezetimibe over-encapsulated tablets orally QD added to stable LMT for 24 weeks.
Ezetimibe 10 mg + Atorvastatin 40 mgACTIVE_COMPARATORParticipants, who were receiving atorvastatin 40 mg over-encapsulated tablets orally at baseline, received ezetimibe 10 mg over-encapsulated tablets orally QD, atorvastatin 40 mg over-encapsulated tablets orally QD, and placebo for alirocumab SC injection Q2W added to stable LMT for 24 weeks.
Alirocumab 75 mg/ up to 150 mg + Atorvastatin 40 mgEXPERIMENTALParticipants, who were receiving atorvastatin 40 mg over-encapsulated tablets orally at baseline, received alirocumab 75 mg SC injection Q2W, atorvastatin 40 mg over-encapsulated tablets orally QD, and placebo for ezetimibe over-encapsulated tablets orally QD added to stable LMT for 24 weeks. Alirocumab dose up-titrated to 150 mg Q2W from Week 12 when LDL-C levels ≥70 mg/dL (1.81 mmol/L) or ≥100 mg/dL (2.59 mmol/L) at Week 8, based on baseline disease characteristic and medical history.
Atorvastatin (statin rechallenge arm)OTHERAtorvastatin 20 mg over-encapsulated tablets orally once daily (QD) for 24 weeks and placebo (for alirocumab) subcutaneous (SC) injection every two weeks (Q2W) for 24 weeks added to stable lipid-modifying therapy (LMT).
EzetimibeACTIVE_COMPARATOREzetimibe 10 mg over-encapsulated tablets orally QD for 24 weeks and placebo (for alirocumab) SC injection Q2W for 24 weeks added to stable LMT.
Alirocumab 75 mg/ up to 150 mgEXPERIMENTALAlirocumab 75 mg SC injection Q2W for 24 weeks and placebo (for atorvastatin/ezetimibe) over-encapsulated tablets orally QD for 24 weeks added to stable LMT. Alirocumab dose up-titrated to 150 mg Q2W from Week 12 when LDL-C levels ≥70 mg/dL (1.81 mmol/L) or ≥100 mg/dL (2.59 mmol/L) at Week 8, based on cardiovascular risk.
Alirocumab and CemiplimabEXPERIMENTALCombination of anti-PCSK9 antibody alirocumab with the anti-PD-1 antibody cemiplimab
Arm 1 - Low DoseEXPERIMENTAL -
Arm 2 - Medium DoseEXPERIMENTAL -
Arm 3 - High DoseEXPERIMENTAL -
Arm 4 - Control DoseEXPERIMENTAL -
Placebo Matched to AlirocumabPLACEBO_COMPARATORParticipants who received placebo in parent study (NCT01576484), has received a subcutaneous injection of placebo matched to alirocumab every 2 weeks for 4 years in this study.
Alirocumab 150 mgEXPERIMENTALParticipants who received alirocumab in parent study (NCT01576484), has received a subcutaneous injection of alirocumab 150 milligram (mg) every 2 weeks for 4 years in this study.
GOFm PCSK9 (Cohort 1): Alirocumab From Day 1EXPERIMENTALParticipants with gain-of-function mutation (GOFm) in proprotein convertase subtilisin/kexin type 9 (PCSK9) gene (Cohort 1): Alirocumab 150 mg subcutaneous (SC) injection at Week 0 (Day 1), Week 2 (Day 15), Week 4, 6 and 10 (matching placebo at Week 8, 12 and 14) during the double-blind period (Group A). Afterwards, participants have the possibility to continue in an open-label extension period with 150 mg alirocumab SC twice per week (Q2W) for an additional 3 years.
GOFm PCSK9 (Cohort 1): Alirocumab From Day 15EXPERIMENTALParticipants with gain-of-function mutation (GOFm) in proprotein convertase subtilisin/kexin type 9 (PCSK9) gene (Cohort 1): Alirocumab 150 mg subcutaneous (SC) injection at Week 2 (Day 15), Week 4, 6, 8 and 12 (\[matching placebo at Week 0 (Day 1), 10 and 14\]) during the double-blind period (Group B). Afterwards, participants have the possibility to continue in an open-label extension period with 150 mg alirocumab SC twice per week (Q2W) for an additional 3 years.
GOFm PCSK9 or LOFm ApoB (Cohort 2): Alirocumab from Day 1EXPERIMENTALParticipants with gain-of-function mutation (GOFm) in the proprotein convertase subtilisin/kexin type 9 (PCSK9) gene or loss-of-function mutation (LOFm) in the apolipoprotein (Apo) B gene (Cohort 2): Alirocumab 150 mg subcutaneous (SC) injection at Week 0 (Day 1), Week 2 (Day 15), Week 4, 6 and 10 (matching placebo at Week 8, 12 and 14) during the double-blind period (Group C). Afterwards, participants have the possibility to continue in an open-label extension period with 150 mg alirocumab SC twice per week (Q2W) for an additional 3 years.
GOFm PCSK9 or LOFm ApoB (Cohort 2): Alirocumab from Day 15EXPERIMENTALParticipants with gain-of-function mutation (GOFm) in proprotein convertase subtilisin/kexin type 9 (PCSK9) gene or loss-of-function mutation (LOFm) in apolipoprotein (Apo) B gene (Cohort 2): Alirocumab 150 mg subcutaneous (SC) injection at Week 2 (Day 15), Week 4, 6, 8 and 12 (\[matching placebo at Week 0 (Day 1), 10 and 14\]) during the double-blind period (Group D). Afterwards, participants have the possibility to continue in an open-label extension period with 150 mg alirocumab SC twice per week (Q2W) for an additional 3 years.
Alirocumab 150 mg Q4WEXPERIMENTALAlirocumab 150 mg SC injection Q4W added to stable statin regimen with or without concomitant ezetimibe for 12 weeks.
Alirocumab 200 mg Q4WEXPERIMENTALAlirocumab 200 mg SC injection Q4W added to stable statin regimen with or without concomitant ezetimibe for 12 weeks.
Alirocumab 300 mg Q4WEXPERIMENTALAlirocumab 300 mg SC injection Q4W added to stable statin regimen with or without concomitant ezetimibe for 12 weeks.
Alirocumab 150 mg Q2WEXPERIMENTALAlirocumab 150 mg SC injection Q2W added to stable statin regimen with or without concomitant ezetimibe for 12 weeks.
AlirocumabEXPERIMENTALCritically ill participants with sepsis leading to cardiovascular and/or respiratory failure who are randomized to receive alirocumab.

Interventions

NameTypeDescription
AlirocumabDRUGAlirocumab SC Q2W
PlaceboDRUGMatching placebo SC Q2W
Placebo (for alirocumab)DRUGSolution for injection, subcutaneous injections in the abdomen, thigh, or outer area of upper arm with an auto-injector.
StatinDRUGAtorvastatin, rosuvastatin and simvastatin at stable dose in participants with stable statin therapy
LMT (atorvastatin, simvastatin, or rosuvastatin)DRUG -
RosuvastatinDRUGRosuvastatin over-encapsulated tablets orally.
EzetimibeDRUGEzetimibe over-encapsulated tablets orally.
AtorvastatinDRUGAtorvastatin over-encapsulated tablets orally.
Alirocumab and CemiplimabCOMBINATION_PRODUCTCombination of PCSK9 inhibitor Alirocumab 150mg SC q2weeks and PD-I inhibitor Cemiplimab 350mg IV q3 weeks
Placebo Matched to AlirocumabDRUGPlacebo matched to alirocumab was supplied in a pre-filled syringe and administered subcutaneously (SC) in the abdomen, thigh, or outer upper arm; REGN727(SAR236553) is an anti-PCSK9 (proprotein convertase subtilisin/kexin type 9) antibody
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Eligibility Criteria

Age Range18 Years to N/A
SexALL
Healthy VolunteersNo
Study Sites28

Note: The information listed below is not intended to contain all considerations relevant to a patient's potential participation in this clinical trial, therefore not all inclusion/exclusion criteria are listed. Key Inclusion Criteria 1. Diagnosis of HoFH by at least 1 of the following genotype or...

Countries:United StatesAustriaCanadaCzechiaFranceGermanyGreeceItalyJapanSouth AfricaTaiwanTurkey (Türkiye)UkraineBulgariaHungaryIsraelNorwaySlovakiaUnited KingdomNetherlandsAustraliaMexicoSpain
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Recent Changes (Last 90 Days)

LOWJul 22, 2026NCT07477704lastUpdatePostDate: changed
LOWJul 22, 2026NCT07477704lastUpdatePostDate: changed
LOWJul 8, 2026NCT07477704lastUpdatePostDate: changed
LOWJul 8, 2026NCT07477704lastUpdatePostDate: changed
LOWJul 2, 2026NCT07477704startDate: changed
LOWJul 2, 2026NCT07477704startDate: changed
LOWJul 2, 2026NCT07477704startDate: changed
LOWJul 2, 2026NCT07477704startDate: changed

Frequently asked questions about Alirocumab

What is Alirocumab used for?

Alirocumab is used to treat high cholesterol conditions, including heterozygous familial hypercholesterolemia, homozygous familial hypercholesterolemia, and hypercholesterolemia. It is also being studied in non-small cell lung cancer and sepsis. It is a monoclonal antibody developed by Regeneron Pharmaceuticals, Inc. (REGN).

What does Alirocumab target?

Alirocumab targets PCSK9 and PD-1. As a monoclonal antibody, it binds these molecular targets, which underlies its use in cholesterol disorders and its investigation in non-small cell lung cancer. The dual PCSK9 and PD-1 targeting reflects both metabolic and immuno-oncology applications.

Who makes Alirocumab?

Alirocumab is developed by Regeneron Pharmaceuticals, Inc., which trades under the ticker REGN. Regeneron is the company behind the drug's clinical development across its cholesterol and oncology programs.

What phase is Alirocumab in?

Alirocumab is in Phase 3 development. It is an investigational monoclonal antibody and is not described as approved. Its clinical program spans Phase 1 through Phase 3 studies, with 8 total trials, of which 7 are completed and 1 is active.

What clinical trials is Alirocumab in?

Alirocumab trials include NCT07477704, a Phase 2 study in hypercholesterolemia that is recruiting; NCT05553834, a Phase 2 study in metastatic non-small cell lung cancer that is active but not recruiting; NCT05469347, a completed Phase 1 sepsis study; and NCT03156621, a completed Phase 3 study in homozygous familial hypercholesterolemia.

Is Alirocumab the same as Cemiplimab?

Alirocumab is also known as Alirocumab and Cemiplimab. People searching for either name are looking at the same Regeneron asset, which targets both PCSK9 and PD-1. Cemiplimab is the PD-1 directed component referenced in this combined naming.