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Glatiramer

Phase 3

Multiple Sclerosis | Small molecule | Neurology |Teva Pharmaceutical Industries Limited|Last Updated: Aug 3, 2022

Success Probability

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Market & Valuation

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Trial Design

RandomizedDouble-BlindPLACEBO_CONTROLLEDDMCBiomarker
Total Trials2
Total Enrollment629

FDA Designations

No designations recorded

Clinical trial landscape

Glatiramer · 9 trials · 5 indications

Phase 3 6Phase 2 3
NCT01167426Evaluation of Two Glatiramer Acetate (GA) Formulations in Relapsing-Remitting Multiple Sclerosis (RRMS) PatientsMultiple Sclerosis
COMPLETED148 Analytics
NCT01067521A Study in Subjects With Relapsing-Remitting Multiple Sclerosis (RRMS) to Assess the Efficacy, Safety and Tolerability of Glatiramer Acetate (GA) Injection 40 mg Administered Three Times a Week Compared to PlaceboRelapsing Remitting Multiple Sclerosis
COMPLETED1,404 Analytics
NCT00947752Safety of New Formulation of Glatiramer AcetateRelapsing Remitting Multiple Sclerosis
COMPLETED147 Analytics
NCT00856635A Randomized, Double-blind, Placebo-controlled, Multicenter Study of the Effects of Glatiramer Acetate (GA) on the Retinal Nerve Fiber Layer (RNFL) and Visual Function in Patients With a First Episode of Acute Optic Neuritis (AON). (Octagon)Optic Neuritis
COMPLETED44 Analytics
NCT00337779Clinical Trial Comparing Treatment of Relapsing-Remitting Multiple Sclerosis (RR-MS) With Two Doses of Glatiramer Acetate (GA).Relapsing Remitting Multiple Sclerosis
COMPLETED1,155 Analytics
NCT00666224Evaluate Early Glatiramer Acetate Treatment in Delaying Conversion to Clinically Definite Multiple Sclerosis of Subjects Presenting With Clinically Isolated SyndromeMultiple Sclerosis
COMPLETED481 Analytics
PHASE3COMPLETED
Evaluation of Two Glatiramer Acetate (GA) Formulations in Relapsing-Remitting Multiple Sclerosis (RRMS) Patients
Multiple SclerosisUnlock trial analytics
PHASE3COMPLETED
A Study in Subjects With Relapsing-Remitting Multiple Sclerosis (RRMS) to Assess the Efficacy, Safety and Tolerability of Glatiramer Acetate (GA) Injection 40 mg Administered Three Times a Week Compared to Placebo
Relapsing Remitting Multiple SclerosisUnlock trial analytics
PHASE3COMPLETED
Safety of New Formulation of Glatiramer Acetate
Relapsing Remitting Multiple SclerosisUnlock trial analytics
PHASE3COMPLETED
A Randomized, Double-blind, Placebo-controlled, Multicenter Study of the Effects of Glatiramer Acetate (GA) on the Retinal Nerve Fiber Layer (RNFL) and Visual Function in Patients With a First Episode of Acute Optic Neuritis (AON). (Octagon)
Optic NeuritisUnlock trial analytics
PHASE3COMPLETED
Clinical Trial Comparing Treatment of Relapsing-Remitting Multiple Sclerosis (RR-MS) With Two Doses of Glatiramer Acetate (GA).
Relapsing Remitting Multiple SclerosisUnlock trial analytics
PHASE3COMPLETED
Evaluate Early Glatiramer Acetate Treatment in Delaying Conversion to Clinically Definite Multiple Sclerosis of Subjects Presenting With Clinically Isolated Syndrome
Multiple SclerosisUnlock trial analytics

Study Endpoints

Primary Endpoints

Change From Week 2 to Week 6 in Composite Score of Patient Satisfaction With Injection Experience
Week 2 (prior to first injection with 20 mg/0.5 mL formulation), Week 6 (after 4 weeks of treatment with 20 mg/0.5 mL formulation).

The Satisfaction with Injection Experience questionnaire consists of 5 questions where participants are asked to rate their injection experience over the past 2 weeks on ease of use, bother, acceptability, confidence to inject and satisfaction. The response options range from "strongly disagree" (score = 1) to "strongly agree" (score = 5). The composite score of Satisfaction with Injection Experience is defined as the mean of the five Likert questions. The composite score ranges from 1.0 to 5.0, with a score of 5.0 representing the most satisfaction with injection experience and a score of 1.0 representing the least satisfaction with injection experience.

Total Number of Confirmed Relapses During the Placebo Controlled (PC) Treatment Period Estimated by Negative Binomial Regression
Day 1 to 12 months

Relapses were monitored throughout the study. During the PC Period, two neurologists/physicians assessed subjects' general medical and neurological evaluations separately. A relapse was defined as the appearance of 1+ new neurological abnormalities or the reappearance of 1+ previously observed neurological abnormalities lasting \>= 48 hours and immediately preceded by an improving neurological state of at \>=30 days from onset of previous relapse. An event was counted as a relapse only when the subject's symptoms were accompanied by observed objective neurological changes, consistent with \>= one of the following: - An increase of \>= 0.5 in the Expanded Disability Status Scale (EDSS) score as compared to previous evaluation. - An increase of one grade in the actual score of \>=2 of the 7 functional systems (FS), as compared to previous evaluation. - An increase of 2 grades in the actual score of one FS as compared to the previous evaluation. Adjusted mean values are displayed.

Annualized Rate of Confirmed Relapses Comparing Early Starters to Delayed Starters Estimated by Negative Binomial Regression
Day 1 up to 6.5 years

The annualized relapse rate (ARR) was calculated for the study by dividing the cumulative number of confirmed relapses by the number of person-years of exposure to treatment. The analysis of the annualized relapse rate is based on estimating a contrast (early start vs delayed start) derived from a baseline-adjusted, Negative Binomial Regression model to the number of confirmed relapses observed during study (post randomization) with an "offset" based on the log of exposure to treatment.

Subject-reported Pain Associated Immediately After Each Injection
5 weeks of injections

A visual analog scale (VAS) was used for subjective characteristics that cannot be directly measured. Respondents specified their level of agreement to a statement by indicating a position along a continuous line between two end-points. The VAS scale used 0 mm to represent "no pain" and up to 100 mm to represent "worst possible pain;" subjects drew a continuous line to represent their level of pain.

Retinal Nerve Fiber Layer Thickness at Baseline and Month 6
Baseline and Month 6

Axonal loss in the optic nerve (due to optic neuritis) was assessed by measuring retinal nerve fiber thickness of the affected eye using optical coherence tomography (OCT) at Baseline and Month 6.

The Rate of Confirmed Relapses During the Double-blind Phase (12 Months).
12 months

A confirmed relapse is defined as the appearance of one or more new neurological abnormalities or the reappearance of one or more previously observed neurological abnormalities. This change in clinical state must last at least 48 hours and be immediately preceded by an improving neurological state of at least thirty (30) days from onset of previous relapse.

Time to Clinically Definite Multiple Sclerosis (CDMS) Conversion
up to 3 years

Data from interim analysis with database lock on October 14, 2007. The time from randomization to conversion to CDMS as determined by the occurrence of a second clinical attack during the double-blind period. Poser criteria are: Two relapses and clinical evidence of two separate lesions; clinical evidence of one lesion and paraclinical evidence of another separate lesion. The two relapses must involve different parts of Central Nervous System and must be separated by a period of at least one month. Lesions are determined by Magnetic Resonance Imaging (MRI).

Twenty-fifth Percentile (25%) Kaplan-Meier Estimates for Time From Randomization to Conversion to Clinically Definite Multiple Sclerosis (CDMS) During the Double-blind Period
up to 3 years

Data from interim analysis with database lock on October 14, 2007. Due to the number of participants in the glatiramer acetate group that converted to CDMS (see outcome #6), the 25th percentile was considered when running the Kaplan-Meier estimate for time to conversion to CDMS. Conversion to CDMS is determined by the occurrence of the second clinical attack.

Slope of Change From Baseline in the ALS Functional Rating Scale (ALSFRS-R)
Baseline, Weeks 4, 8, 12, 17, 22, 26, 31, 36, 40, 44, 48, 52

The ALSFRS-R is a questionnaire-based scale for monitoring the progression of disability in patients with ALS. It is composed of 12 items, each scored between 0 and 4.The total score, calculated as the sum of these 12 items, ranges from 0 to 48. The higher the score, the less disabled the participant. Timepoints after baseline were included in calculation of slope of change in ALSFRS-R. Slope is derived from the time by treatment interaction term from the Repeated Measures Analysis of Covariance model. Descriptive statistics of the slope are reported.

The total number of T1 Gd-enhancing lesions in T1-weighted images, as measured at months 7, 8 and 9
Determine if short-term immunosuppression with mitoxantrone (Novantrone®) followed by chronic treatment with Glatiramer Acetate (GA) in comparison to treatment with GA for the same period of time but without immunosuppression is well-tolerated and safe
15 months

Secondary Endpoints

Patient Injection Experience Preference
Week 4
The Cumulative Number of New/Enlarging T2 Lesions Taken at Month 6 and Month 12 During the Placebo Controlled (PC) Treatment Period Estimated by Negative Binomial Regression
Baseline (Day -7), Month 6, Month 12
The Cumulative Number of Gadolinium (Gd)-Enhanced Lesions on T1-Weighted Images At Month 6 and Month 12 of the Placebo-Controlled (PC) Treatment Period Estimated by Negative Binomial Regression
Baseline (Day -7), Month 6, Month 12
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Study Design & Arms

AllocationNON_RANDOMIZED
MaskingNONE
ModelSINGLE_GROUP

Treatment Arms

ArmTypeDescription
20 mg/0.5 mL Glatiramer AcetateACTIVE_COMPARATORParticipants received once daily subcutaneous administration of 20 mg glatiramer acetate as 20 mg/1.0 mL utilizing autoject 2 for glass syringe for two weeks (Period 1), followed by 20 mg/0.5 mL utilizing the autoject 2 device for four weeks (Period 2).
GA 40 mg / GA 40 mgEXPERIMENTALAlso referred to as the 'Early Start' treatment arm, participants were administered glatiramer acetate (GA) 40 mg/mL by subcutaneous injection three times a week for 12 months during the Double-Blind Period, and then continued that treatment as open-label therapy until the drug was commercially available or development stopped.
Placebo / GA 40 mgPLACEBO_COMPARATORAlso referred to as the 'Delayed Start' treatment arm, participants were administered placebo subcutaneous injections three times a week for 12 months during the Double-Blind Period, and then switched to GA 40 mg/mL subcutaneous injections three times a week as open-label therapy until the drug was commercially available or development stopped.
F1 Glatiramer acetate 20mg/1.0mlACTIVE_COMPARATOR -
F2 Glatiramer acetate 20mg/0.5mlEXPERIMENTAL -
Glatiramer acetateEXPERIMENTALParticipants received glatiramer acetate 20 mg subcutaneous injection once a day for up to 6 months.
PlaceboPLACEBO_COMPARATORParticipants received placebo subcutaneous injection once a day for up to 6 months.
glatiramer acetate 40 mgACTIVE_COMPARATOR -
glatiramer acetate 20 mgACTIVE_COMPARATOR -
Placebo (DB) to GA (OL)PLACEBO_COMPARATORPlacebo matching glatiramer acetate once daily by subcutaneous injection during the double-blind period (DB). Glatiramer acetate (GA) 20 mg once daily by subcutaneous injection during the open-label period (OL).
40 mg glatiramer acetate (GA)EXPERIMENTALPre-filled syringe of 40 mg glatiramer acetate (GA) for injection, administered subcutaneously once a day.
Copaxone 20 mgACTIVE_COMPARATORCopaxone 20 mg
Copaxone 20mg with Novantrone inductionACTIVE_COMPARATORCopaxone 20mg with Novantrone induction

Interventions

NameTypeDescription
Glatiramer Acetate 20 mg/0.5 mLDRUG20 mg/1.0 mL formulation of glatiramer acetate utilizing the autoject 2 for glass syringe.
Glatiramer acetate (GA)DRUGGA 40 mg/mL administered 3 times a week by subcutaneous injection for a period of 12 months for participants assigned to GA treatment in the Double-Blind Period, and GA 40 mg/mL administered 3 times a week by subcutaneous injection for all participants in the Open-Label Extension Period.
PlaceboDRUGPlacebo comparator administered by subcutaneous injection three times each week for 12 months during the Double-Blind Period.
Glatiramer AcetateDRUGSubjects received both doses once daily in a crossover fashion, for a total treatment duration of five weeks, including a one-week run-in period. Subject-reported injection pain was recorded in a daily diary.
Experimental Glatiramer AcetateDRUGGA 20 mg/0.5 mL
Glatiramer Acetate (GA) 40 mgDRUGGlatiramer Acetate Injection 40 mg/ml Daily subcutaneous injection for 12 months
glatiramer acetate 20 mgDRUGGlatiramer Acetate Injection 20 mg/ml Daily subcutaneous injection for 12 months
Glatiramer Acetate (DB)DRUGDouble blind period (DB): glatiramer acetate (GA) by subcutaneous injection, 20mg, once daily, for up to 36 months or until conversion to clinically definite multiple sclerosis (CDMS).
Glatiramer Acetate (OL)DRUGOpen label period (OL): glatiramer acetate (GA), 20 mg, subcutaneous injection, once daily, given for up to an additional 24 months.
40 mg glatiramer acetateDRUGparenteral drug
glatiramer acetate 40 mgDRUGglatiramer acetate 40 mg
glatiramer acetate 20 mg, with mitoxantroneDRUGglatiramer acetate 20 mg, with mitoxantrone
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Eligibility Criteria

Age Range18 Years to N/A
SexALL
Healthy VolunteersNo

Inclusion Criteria: * Patients ≥ 18 years of age with a diagnosis of Relapse Remitting Multiple Sclerosis (RRMS) or Clinically Isolated Syndrome (CIS) * Currently injecting glatiramer acetate 20 mg/1.0 mL per day subcutaneously (SC) for a minimum of 90 days utilizing the autoject 2 for glass syring...

Countries:United StatesBulgariaCroatiaCzechiaEstoniaGeorgiaGermanyHungaryIsraelItalyLithuaniaPolandRomaniaRussiaSouth AfricaUkraineUnited KingdomBelgiumFrance
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Frequently asked questions about Glatiramer

What is Glatiramer used for?

Glatiramer is used for multiple sclerosis, specifically relapse-remitting multiple sclerosis, as well as optic neuritis and amyotrophic lateral sclerosis. It is being studied in clinical trials for these neurological conditions. The drug is developed by Teva Pharmaceutical Industries Limited.

What does Glatiramer target?

Glatiramer targets the immune system in multiple sclerosis. It is a small molecule that modulates immune responses, though the exact molecular target is not specified. It is being investigated for its effects on relapse-remitting multiple sclerosis and other neurological conditions.

Who makes Glatiramer?

Glatiramer is developed by Teva Pharmaceutical Industries Limited, a company traded under the ticker TEVA. Teva is conducting clinical trials to evaluate the drug's effectiveness and safety for multiple sclerosis and related conditions.

What phase is Glatiramer in?

Glatiramer is in Phase 3 clinical development. It has completed four trials, including Phase 3 studies for multiple sclerosis and optic neuritis. The drug is investigational and not yet approved, as it is still undergoing clinical evaluation.

What clinical trials is Glatiramer in?

Glatiramer has completed four clinical trials, including NCT00202982, NCT00666224, NCT00856635, and NCT01167426. These trials studied the drug in relapse-remitting multiple sclerosis, clinically isolated syndrome, optic neuritis, and multiple sclerosis, with a total enrollment of 2,746 participants.

Is Glatiramer the same as Copaxone?

Glatiramer is the active ingredient in Copaxone, a brand name for the drug. Clinical trials have tested higher doses of Copaxone, such as 40mg, compared to the approved 20mg dose. Glatiramer acetate is the full name of the compound.