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Immutep Announces Positive Update from Phase I Study of IMP761, a First-in-Class LAG-3 Agonist Antibody for Autoimmune Diseases Initial pharmacological data from placebo-controlled, double-blind Phase I study shows signi

Key Takeaway: Immutep Limited has reported positive initial efficacy and safety data from a Phase I study of its first-in-class LAG-3 agonist antibody, IMP761, aimed at treating autoimmune diseases. The study, which is double-blind and placebo-controlled, indicates substantial T cell suppression at the highest dose without any treatment-related adverse events. Given these encouraging results, Immutep plans to continue exploring higher dosing levels. The ongoing trial is being conducted in the Netherlands, and further data is expected to be released in late 2025.

Market Sentiment Analysis

POSITIVE FACTORS

  • Initial efficacy data from the Phase I study is promising.
  • No treatment-related adverse events reported in healthy participants.
  • Significant T cell suppression observed at the highest dosing level.
  • The targeted approach of IMP761 may result in fewer side effects.

Full Press Release Details

Immutep Announces Positive Update from Phase I Study of IMP761,
LAG-3 Agonist Antibody for Autoimmune Diseases
SYDNEY, AUSTRALIA June 23, 2025 Immutep Limited (ASX: IMM; NASDAQ: IMMP) ( Immutep or
the Company ), a late-stage immunotherapy company targeting cancer and autoimmune diseases, today announces positive initial efficacy data and continued favourable safety data from the placebo-controlled, double-blind first-in-human Phase I study evaluating IMP761, a first-in-class
LAG-3 agonist antibody for autoimmune diseases.
Through the highest dosing level to date (0.9 mg/kg of IMP761),
there have been no treatment-related adverse events in healthy participants. Additionally, pharmacodynamic data at this dosing level show that the inhibition of T cell infiltration in the skin at day 10 following a neoantigen rechallenge has already
reached 80%. Given the encouraging efficacy and safety, Immutep is continuing with single ascending dose levels of 2.5, 7 and 14 mg/kg.
The LAG-3 (lymphocyte-activation gene-3) immune checkpoint has been identified as a promising therapeutic target for rheumatoid arthritis, Type 1 diabetes, and multiple sclerosis
in multiple publications.1-3 IMP761 is the first LAG-3 agonist antibody developed to potentially treat these large and growing disorders, each of which
represent multi-billion dollar markets, and many other autoimmune diseases. By enhancing the brake function of LAG-3 to silence dysregulated self-antigen-specific memory T cells, IMP761 is designed
to target the cause of autoimmune diseases and restore balance to the immune system.
Dr. Fr d ric Triebel, CSO of
Immutep, said: The early pharmacological data showing substantial T cell suppression at the highest dose level of IMP761 are very promising, especially in conjunction with its continued favourable safety profile, and highlight the
potential efficacy of this LAG-3 agonist in treating autoimmune diseases. LAG-3 expression on activated T cells is known to be highly specific to disease sites, and
particularly in areas of chronic inflammation. This unique specificity enables the potential for IMP761 to have a more targeted approach with fewer side effects than other therapies. We look forward to evaluating higher dosing levels of IMP761 and
hope to further enhance its ability to safely silence the dysregulated T cells responsible for many autoimmune diseases.
The Phase I trial is being
conducted by the Centre for Human Drug Research (CHDR) in Leiden, the Netherlands. In addition to the safety analysis, CHDR is implementing its keyhole limpet haemocyanin (KLH) challenge model to evaluate IMP761 s pharmacological activity.
Additional data from the Phase I to follow in second half of CY2025. For more information on the trial, please visit clinicaltrials.gov (NCT06637865).
IMP761, a first-in-class immunosuppressive lymphocyte-activation gene-3 (LAG-3) agonist antibody, has the
potential to address the root cause of many autoimmune diseases by specifically silencing autoimmune memory T cells that accumulate at disease sites and restoring balance to the immune system. As published in the Journal of Immunology,
encouraging pre-clinical in vivo and in vitro studies show IMP761 inhibits peptide-induced T cell proliferation, activation of human primary T cells, and an antigen-specific delayed-type hypersensitivity (DTH)
reaction.4 Additional preclinical data in oligoarticular juvenile idiopathic arthritis (o-JIA) published in Pediatric Research details how IMP761 led
to a decrease in a broad spectrum of effector cytokines.5 This study also shows children with o-JIA have a skewed
LAG-3 metabolism and suggests they can benefit from agonistic LAG-3 activity.
Immutep is a late-stage biotechnology
company developing novel immunotherapies for cancer and autoimmune diseases. The Company is a pioneer in the understanding and advancement of therapeutics related to Lymphocyte Activation Gene-3 (LAG-3), and its diversified product portfolio harnesses LAG-3 s ability to stimulate or suppress the immune response. Immutep is dedicated to leveraging its expertise to
bring innovative treatment options to patients in need and to maximise value for shareholders. For more information, please visit www.immutep.com.
Australian Investors/Media:
Chris Basta, VP, Investor Relations and
Corporate Communications
+1 (631) 318 4000; chris.basta@immutep.com
This announcement was authorised for release by the CEO of Immutep Limited.
Immutep Limited, Level 32, Australia Square, 264 George Street, Sydney NSW 2000, Australia

Frequently Asked Questions

What is IMP761 developed for?

IMP761 is developed as a LAG-3 agonist antibody for autoimmune diseases.

What were the results of the Phase I study for IMP761?

The Phase I study showed promising efficacy data and a favorable safety profile.

What is LAG-3's role in autoimmune diseases?

LAG-3 helps silence dysregulated memory T cells, targeting autoimmune disease causes.

Who is conducting the Phase I trial for IMP761?

The Centre for Human Drug Research (CHDR) in the Netherlands is conducting the trial.

When can we expect more data from the IMP761 trial?

Additional data from the Phase I trial is expected in the second half of 2025.

Last updated: Jun 23, 2025