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Ponezumab

Phase 2

Cerebral Amyloid Angiopathy | Monoclonal antibody | Neurology |Pfizer, Inc.|Last Updated: May 10, 2017

Success Probability

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Trial Design

RandomizedDouble-BlindPLACEBO_CONTROLLED
Total Trials1
Total Enrollment36

FDA Designations

No designations recorded

Clinical trial landscape

Ponezumab · 1 trial · 1 indication

Phase 2 1
NCT01821118Study Evaluating the Safety,Tolerability and Efficacy of PF-04360365 in Adults With Probable Cerebral Amyloid AngiopathyCerebral Amyloid Angiopathy
COMPLETED36 Analytics
PHASE2COMPLETED
Study Evaluating the Safety,Tolerability and Efficacy of PF-04360365 in Adults With Probable Cerebral Amyloid Angiopathy
Cerebral Amyloid AngiopathyUnlock trial analytics

Study Endpoints

Primary Endpoints

Change From Baseline to Day 2 in Cerebrovascular Reactivity as Measured by the Slope (Amplitude Over Time to Peak) From Visual Task-evoked Functional Magnetic Resonance Imaging (fMRI)
Baseline, Day 2

Blood Oxygen Level Dependant (BOLD) fMRI was performed at Screening (Baseline) and on Days 2 and 90. During each of these sessions, BOLD fMRI images were acquired in rapid succession as a flashing radial black and white checkerboard was presented alternately with a gray screen. This well established visual stimulus is known to produce a reliable increase in BOLD fMRI signal within the visual cortex region of the occipital lobe. The time course of the BOLD fMRI signal was used to assess the vascular reactivity. Imaging sites also acquired cerebral blood flow data using Arterial Spin Labeled (ASL) scans at Screening and on Days 2 and 90. A standard T1-weighted image was also acquired to aid image analysis. All efficacy scans were analyzed centrally. Geometric means are presented in the original scale and standard errors (SE) are presented in logarithmic (log e) scale.

Change From Baseline to Day 90 in Cerebrovascular Reactivity as Measured by the Slope (Amplitude Over Time to Peak) From Visual Task-evoked fMRI
Baseline, Day 90

BOLD fMRI was performed at Screening (Baseline) and on Days 2 and 90. During each of these sessions, BOLD fMRI images were acquired in rapid succession as a flashing radial black and white checkerboard was presented alternately with a gray screen. This well established visual stimulus is known to produce a reliable increase in BOLD fMRI signal within the visual cortex region of the occipital lobe. The time course of the BOLD fMRI signal was used to assess the vascular reactivity. Imaging sites also acquired cerebral blood flow data using ASL scans at Screening and on Days 2 and 90. A standard T1-weighted image was also acquired to aid image analysis. All efficacy scans were analyzed centrally. Geometric means are presented in the original scale and SEs are presented in log e scale.

Secondary Endpoints

Change From Baseline to Day 2 and Day 90 in Cerebrovascular Reactivity as Measured by the Time to Peak From Visual Task-evoked fMRI
Baseline, Day 2, Day 90
Change From Baseline to Day 2 and Day 90 in Cerebrovascular Reactivity as Measured by the Amplitude From Visual Task-evoked fMRI
Baseline, Day 2, Day 90
Change From Baseline to Day 2 and Day 90 in Cerebrovascular Reactivity as Measured by the Time to Return to Baseline From Visual Task-evoked fMRI
Baseline, Day 2, Day 90
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Study Design & Arms

AllocationRANDOMIZED
MaskingTRIPLE
ModelPARALLEL
PurposeTREATMENT

Treatment Arms

ArmTypeDescription
1EXPERIMENTAL -
2PLACEBO_COMPARATOR -

Interventions

NameTypeDescription
PonezumabBIOLOGICALInfusion of Ponezumab (Day 1=10mg/kg; Day 30 and Day 60 dose = 7.5mg/kg) or placebo (saline); administered via infusion for a total infusion time of 20 minutes.
placeboOTHERplacebo (saline)- given via infusion total infusion time of 20 minutes
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Eligibility Criteria

Age Range55 Years to 90 Years
SexALL
Healthy VolunteersNo
Study Sites13

Inclusion Criteria: * Patients diagnosed with probable CAA using the Boston criteria; with no clinical cognitive impairment * In general good health Exclusion Criteria: * Co-morbid diagnosis of clinically documented Alzheimer's disease or significant cognitive impairment * Clinically significant ...

Countries:United StatesCanadaFranceNetherlandsUnited Kingdom
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Frequently asked questions about Ponezumab

What is Ponezumab used for?

Ponezumab is an investigational monoclonal antibody being studied for the treatment of Cerebral Amyloid Angiopathy, a condition characterized by amyloid deposits in the brain's blood vessels. It is being developed by Pfizer, Inc. for this neurological indication.

What does Ponezumab target?

Ponezumab is a monoclonal antibody designed to target amyloid beta, a protein that accumulates in the brain and is associated with Cerebral Amyloid Angiopathy. By binding to this protein, the drug aims to reduce its deposition and potentially slow disease progression.

Who makes Ponezumab?

Ponezumab is being developed by Pfizer, Inc., a multinational pharmaceutical company listed on the New York Stock Exchange under the ticker symbol PFE. The company is conducting clinical trials to evaluate the drug's safety and efficacy in patients with Cerebral Amyloid Angiopathy.

What phase is Ponezumab in?

Ponezumab is currently in Phase 2 clinical development. It has completed one Phase 2 trial, and it remains an investigational drug, meaning it has not yet been approved by regulatory authorities for commercial use.

What clinical trials is Ponezumab in?

Ponezumab has been evaluated in a Phase 2 clinical trial registered as NCT01821118, which studied the safety, tolerability, and efficacy of the drug in adults with probable Cerebral Amyloid Angiopathy. The trial was completed and enrolled 36 participants across the United States, Canada, France, Netherlands, and the United Kingdom.

Is Ponezumab the same as PF-04360365?

Yes, Ponezumab is also known as PF-04360365. The clinical trial NCT01821118, which evaluated the drug for Cerebral Amyloid Angiopathy, used the name PF-04360365 in its official title, confirming that both names refer to the same investigational monoclonal antibody.