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CF102

Phase 2

Hepatocellular Carcinoma | Small molecule | Oncology |Can-Fite Biopharma Ltd|Last Updated: Oct 4, 2022

Success Probability

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Trial Design

RandomizedDouble-BlindCONTROLLEDDMC
Total Trials2
Total Enrollment97

FDA Designations

No designations recorded

Clinical trial landscape

CF102 · 3 trials · 2 indications

Phase 2 2Phase 1 1
NCT02927314A Study of the Efficacy and Safety of CF102 in the Treatment of Non-Alcoholic Fatty Liver DiseaseNon-alcoholic Steatohepatitis (NASH)
COMPLETED60 Analytics
NCT02128958Phase 2, Randomized, Double-Blind, Placebo-Controlled of the Efficacy and Safety of CF102 in Hepatocellular Carcinoma (HCC)Hepatocellular Carcinoma
COMPLETED78 Analytics
PHASE2COMPLETED
A Study of the Efficacy and Safety of CF102 in the Treatment of Non-Alcoholic Fatty Liver Disease
Non-alcoholic Steatohepatitis (NASH)Unlock trial analytics
PHASE2COMPLETED
Phase 2, Randomized, Double-Blind, Placebo-Controlled of the Efficacy and Safety of CF102 in Hepatocellular Carcinoma (HCC)
Hepatocellular CarcinomaUnlock trial analytics

Study Endpoints

Primary Endpoints

Efficacy of CF102 as determined by change in serum alanine aminotransferase (ALT) levels
12 weeks

Mean percent change in serum alanine aminotransferase (ALT) levels

Efficacy of CF102 as determined by change in magnetic resonance imaging-determined hepatic steatosis
12 weeks

Percent change from Baseline in hepatic steatosis measured by magnetic resonance imaging-determined proton-density fat-fraction (MRI-PDFF)

Number of Subjects With Overall Survival
From date of first treatment (Cycle 1 Day 1) until date of death from any cause, assessed up to 12 months

Evaluate the efficacy of orally administered CF102 25 mg twice daily (BID) as compared to placebo, as determined by Overall Survival (OS), when used as second-line therapy in subjects with advanced hepatocellular carcinoma (HCC) and Child-Pugh Class B (CPB) cirrhosis. Overall Survival is defined as the time from Baseline (Cycle 1 Day 1) to death due to any cause, calculated as (date of death- date of Cycle 1 Day 1) +1.OS will be summarized in months, which will be obtained by dividing OS in days by 30 days/month. Summary statistics will be determined using the Kaplan-Meier (KM) estimate of the survival function and the between-treatment comparison will be performed using the logrank test as the primary analysis.

Dose Limiting Toxicity
From start of treatment until Day 28 of Cycle 1

Dose-limiting toxicity was defined as a clinically significant AE or laboratory abnormality occurring in Cycle 1

Maximum Tolerated Dose
first 28 days (Cycle 1)

The MTD was defined as the highest dose level at which \< 2 of 6 patients developed Cycle 1 DLT.

Maximum Plasma Concentration of CF102 (Cmax)
Dose Escalation Phase on Day 1 and Day 29 pre-dose and at 1, 2, 3, 4, 6, 8 hours post-dose

Blood samples were collected and plasma concentrations determined using a high-pressure liquid chromatography method.

Secondary Endpoints

Body weight in subjects with NAFLD
12 weeks
Waist circumference in subjects with NAFLD
12 weeks
HDL cholesterol levels in subjects with NAFLD
12 weeks
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Study Design & Arms

AllocationRANDOMIZED
MaskingTRIPLE
ModelPARALLEL
PurposeTREATMENT

Treatment Arms

ArmTypeDescription
PlaceboPLACEBO_COMPARATORPlacebo tablets orally q12h
CF102 12.5mgACTIVE_COMPARATORCF102 tablets orally q12h
CF102 25mgACTIVE_COMPARATORCF102 tablets orally q12h
CF102EXPERIMENTALorally q12h
Placebo tablets of CF102PLACEBO_COMPARATORorally q12h
CF102 1mgEXPERIMENTALAn open-label trial in 28-day cycles.
CF102 5mgEXPERIMENTALAn open-label trial in 28-day cycles.

Interventions

NameTypeDescription
CF102DRUGorally q12h
PlaceboDRUGorally q12h
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Eligibility Criteria

Age Range18 Years to N/A
SexALL
Healthy VolunteersNo
Study Sites3

Inclusion Criteria: 1. At least 18 years of age. 2. Diagnosis of NAFLD by non-invasive determination of liver triglyceride concentration, as defined as triglyceride concentration ≥10.0% by NMRS. 3. At least 2 of the following: * Obesity, defined as body mass index (BMI) of ≥25 and ≤40 kg/m2; or...

Countries:IsraelUnited StatesBulgariaRomaniaSerbia
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Frequently asked questions about CF102

What is CF 102 used for?

CF 102 is an investigational small molecule being studied for chronic Hepatitis C, hepatocellular carcinoma (HCC), and non-alcoholic steatohepatitis (NASH). It is being developed by Can-Fite Biopharma Ltd (CANF) and is currently in Phase 1 clinical development for these indications.

What does CF 102 target?

CF 102 is a small molecule that targets the A3 adenosine receptor, which is involved in the regulation of cell growth and apoptosis. By activating this receptor, CF 102 may inhibit tumor cell proliferation and reduce inflammation, making it a potential treatment for liver diseases and cancers.

Who makes CF 102?

CF 102 is being developed by Can-Fite Biopharma Ltd, a biopharmaceutical company traded on the stock exchange under the ticker symbol CANF. The company is focused on developing small molecule drugs for inflammatory and oncological indications.

What phase is CF 102 in?

CF 102 is currently in Phase 1 clinical development. It has completed Phase 1 and Phase 2 trials for hepatocellular carcinoma, chronic Hepatitis C, and non-alcoholic steatohepatitis, but it is not yet approved by regulatory authorities and remains investigational.

What clinical trials is CF 102 in?

CF 102 has completed several clinical trials, including NCT00790218 for advanced hepatocellular carcinoma, NCT00790673 for chronic Hepatitis C genotype 1, NCT02128958 for hepatocellular carcinoma, and NCT02927314 for non-alcoholic fatty liver disease. These trials have been completed, with no active trials currently ongoing.

Is CF 102 the same as other drugs?

CF 102 is also known as namodenoson, a selective A3 adenosine receptor agonist. It is being investigated for its potential to treat liver diseases and cancers, and it is distinct from other adenosine receptor agonists due to its high selectivity for the A3 subtype.