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TQJ230

Phase 3

Cardiovascular Disease and Lipoprotein(a) | Small molecule | Cardiovascular |Novartis AG|Last Updated: Aug 28, 2026

Success Probability

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Market & Valuation

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Trial Design

RandomizedDouble-BlindPLACEBO_CONTROLLEDDMC
Total Trials1
Total Enrollment8,323

FDA Designations

No designations recorded

Clinical trial landscape

TQJ230 · 2 trials · 2 indications

Phase 3 2
NCT06267560Lp(a) Lowering Study of Pelacarsen (TQJ230) in US Black/African American and Hispanic Participants With Elevated Lp(a) and Established ASCVDElevated Lp(a) and Established Atherosclerotic Cardiovascular Disease
COMPLETED422 Analytics
NCT04023552Assessing the Impact of Lipoprotein (a) Lowering With Pelacarsen (TQJ230) on Major Cardiovascular Events in Patients With CVDCardiovascular Disease and Lipoprotein(a)
COMPLETED8,323 Analytics
PHASE3COMPLETED
Lp(a) Lowering Study of Pelacarsen (TQJ230) in US Black/African American and Hispanic Participants With Elevated Lp(a) and Established ASCVD
Elevated Lp(a) and Established Atherosclerotic Cardiovascular DiseaseUnlock trial analytics
PHASE3COMPLETED
Assessing the Impact of Lipoprotein (a) Lowering With Pelacarsen (TQJ230) on Major Cardiovascular Events in Patients With CVD
Cardiovascular Disease and Lipoprotein(a)Unlock trial analytics

Study Endpoints

Primary Endpoints

Change in log-transformed Lp(a) concentration from baseline at week 52
Baseline, week 52

The primary aim of the study is to demonstrate the superiority of pelacarsen to placebo in lowering the Lp(a) level at 12 months of treatment in US Black/African American and US Hispanic participants with established ASCVD and a Lp(a) level of ≥ 125 nmo/L.

Time to first occurrence of clinical endpoint committee confirmed expanded major adverse cardiovascular events in patients with elevated Lp(a) ≥ 70 mg/dL
approximately 4 years

Demonstrate the superiority of pelacarsen (TQJ230) compared to placebo in reducing the risk of expanded MACE (cardiovascular death, non-fatal MI, non-fatal stroke and urgent coronary re-vascularization requiring hospitalization) in the overall study population with established CVD and (Lp(a) ≥ 70 mg/dL)

Time to the first occurrence of clinical endpoint committee confirmed expanded major adverse cardiovascular events in a population of patients with elevated Lp(a) ≥ 90 mg/dL.
approximately 4 years

Demonstrate the superiority of pelacarsen (TQJ230) compared to placebo in reducing the risk of expanded MACE (cardiovascular death, non-fatal MI, non-fatal stroke and urgent coronary re-vascularization requiring hospitalization) in the overall study population with established CVD and (Lp(a) ≥ 90 mg/dL)

Secondary Endpoints

Incidence proportion of study discontinuations due to TEAEs
Up to 52 weeks
Incidence proportion of Treatment emergent adverse events (TEAEs) of special interest
Up to 52 weeks
Time to the first occurrence of the clinical endpoint committee confirmed composite endpoint of major adverse cardiovascular events (CV death, non-fatal MI, and non-fatal stroke)
approximately 4 years
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Study Design & Arms

AllocationRANDOMIZED
MaskingQUADRUPLE
ModelPARALLEL
PurposeTREATMENT

Treatment Arms

ArmTypeDescription
TQJ230EXPERIMENTALTQJ230 80mg QM s.c.
PlaceboPLACEBO_COMPARATORMatching placebo.

Interventions

NameTypeDescription
TQJ230DRUGTQJ230 80mg QM s.c.
PlaceboDRUGmatching placebo
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Eligibility Criteria

Age Range18 Years to 80 Years
SexALL
Healthy VolunteersNo
Study Sites96

Inclusion Criteria: * Male and female US Black/African American and US Hispanic participants 18 to ≤ 80 years of age * Lp(a) ≥ 125 nmol/L at the screening visit, measured at the Central laboratory * On Standard of Care (SoC) therapy for risk factors other than Lp(a), including LDL-C (LDL-C lowering...

Countries:United StatesPuerto RicoArgentinaAustraliaAustriaBelgiumBrazilBulgariaCanadaChileChinaColombiaCzechiaDenmarkFranceGermanyGreeceGuatemalaHong KongHungaryIcelandIndiaIsraelItalyJapanMexicoNetherlandsNorwayPeruPolandPortugalRomaniaRussiaSlovakiaSloveniaSouth AfricaSouth KoreaSpainSwedenSwitzerlandTaiwanTurkey (Türkiye)United Kingdom
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Recent Changes (Last 90 Days)

HIGHAug 28, 2026NCT04023552Status: ACTIVE_NOT_RECRUITING → COMPLETED
HIGHAug 28, 2026NCT04023552Status: ACTIVE_NOT_RECRUITING → COMPLETED
MEDIUMJun 20, 2026NCT06267560TRIAL_REMOVED: changed
MEDIUMJun 20, 2026NCT06267560TRIAL_REMOVED: changed
MEDIUMJun 20, 2026NCT06267560TRIAL_REMOVED: changed

Frequently asked questions about TQJ230

What is TQJ230 used for?

TQJ230, also known as pelacarsen, is an investigational small molecule being studied for cardiovascular disease and elevated lipoprotein(a), specifically in patients with elevated Lp(a) and established atherosclerotic cardiovascular disease. It is in Phase 3 clinical development and is not yet approved by the FDA.

What does TQJ230 target?

TQJ230 targets lipoprotein(a), or Lp(a), a risk factor for cardiovascular disease. By lowering Lp(a) levels, the drug aims to reduce major cardiovascular events in patients with established cardiovascular disease and elevated Lp(a).

Who makes TQJ230?

TQJ230 is being developed by Novartis AG, a multinational pharmaceutical company traded on the New York Stock Exchange under the ticker symbol NVS. The drug is currently in Phase 3 clinical trials for cardiovascular indications.

What phase is TQJ230 in?

TQJ230 is in Phase 3 clinical development. It is an investigational drug, meaning it has not been approved by regulatory authorities. Two Phase 3 trials have been completed, including a large study with over 8,000 participants.

What clinical trials is TQJ230 in?

TQJ230 has been studied in two Phase 3 trials. The first, NCT04023552, enrolled 8,323 patients with cardiovascular disease and elevated Lp(a) across multiple countries. The second, NCT06267560, enrolled 422 US Black/African American and Hispanic participants with elevated Lp(a) and established atherosclerotic cardiovascular disease.

Is TQJ230 the same as pelacarsen?

Yes, TQJ230 is also known as pelacarsen. Both names refer to the same investigational drug developed by Novartis for lowering lipoprotein(a) in patients with cardiovascular disease.